By Jorge Ortiz, Jason André
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Additional info for After the Kidney Transplant - The Patients and Their Allograft
1 Superficial HSV infection Superficial herpes simplex virus (HSV) infection is defined as disease limited to the skin or mucosal surfaces without evidence of dissemination to visceral organs. Serologic evidence of HSV1 and HSV2 is common in the general population. Although periodic reactivation of HSV1 and HSV2 infection occurs, these episodes tend to be selflimited in immunocompetent individuals. , 1985). The highest incidence of HSV reactivation occurs early after transplantation, with the greatest risk occurring during the first month following transplantation (3).
Specific syndromes include the hematologic manifestations of malaria, myocarditis in Chagas’ disease, acute renal failure in malaria and leishmaniasis, and the typical skin lesions of Chagas’ and cutaneous leishmaniasis. Many antiparasitic drugs have the potential for gastrointestinal, hepatic, renal, and hematologic toxicity, and may interact with the metabolism of immunosuppressive agents. It is recommended that transplant clinicians have a high index of suspicion of parasitic infections as an important transmission threat, as well as a potential cause of significant posttransplant morbidity (Barsoum, 2004).
2601-2614. Fishman, JA. & Davis, JA. (2008). Infections in renal transplant recipients. Morris, Stuart J. Knechtle. (eds). KIDNEY TRANSPLANTATION: PRINCIPLES AND PRACTICE, 6th ed. Elsevier, pp. 492-507. ISBN: 978-1-4160-3343-1. Philadelphia. ; Belzer, FO. & Maki, DG. (1990). A prospective, randomized, double-blind study of trimethoprim-sulfamethoxazole for prophylaxis of infection in renal transplantation: Clinical efficacy, absorption of trimethoprimsulfamethoxazole, effects on the microflora, and the cost-benefit of prophylaxis.